Differential Response to Trichloroethylene-Induced Hepatosteatosis in Wild-Type and PPARα-Humanized Mice

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Differential Response to Trichloroethylene-Induced Hepatosteatosis in Wild-Type and PPARα-Humanized Mice

BACKGROUND Trichloroacetic acid, an oxidative metabolite of trichloroethylene (TRI), is a ligand of the peroxisome proliferator-activated receptor alpha (PPAR) alpha, which is involved in lipid homeostasis and anti-inflammation. OBJECTIVE We examined the role of mouse and human PPARalpha in TRI-induced hepatic steatosis and toxicity. METHODS Male wild-type (mPPARalpha), Pparalpha-null, and ...

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ژورنال

عنوان ژورنال: Environmental Health Perspectives

سال: 2010

ISSN: 0091-6765,1552-9924

DOI: 10.1289/ehp.1001928